Thanks Chris, for sharing your story. It is nice to read the progress in your d. It helps me to develop my opinion for my own treatment.
Good luck with your pump and I would love to hear your experiences with the Omnipod. Does everybody with a pump have CGM as well?
Hi Yota, thanks for telling me your story. You are just a few months earlier diagnosed than I was (March 2011). Keep me posted on your situation and good luck to you too!
No, Smile, I have a pump, but no CGM. For right now I don’t really feel the need for a CGM. My control is ok and I can’t really afford the supplies for another piece of equipment, plus wearing one thing on my body is enough. Plus I think I’d obsess and look at it all the time. I’ve never worn a watch for the same reason! But some people swear by their CGMs.
Since T1 is an autoimmune process T2 can not develop into T1. But T2 can loose that many of their beta cells that the treatment is 1:1 with T1. Different causes same outcome.
Metformin is not appropriate for Type 1. I hope you go back to your doctor’s soon.
While metformin isn’t a primary drug for T1s (or 1.5’s), I wouldn’t count it out. It has a variety of uses including inhibiting the Liver’s production of glucose (glucoseneogenesis), which can aid more than just T1s. It also has uses for polycystic ovarian syndrome (related to insulin resistance) by increasing insulin resistance. It also reduces the uptake of glucose in the GI tract.
That being said…it shouldn’t be simply given to everyone and may have little/no impact. It should be avoided or used very carefully for those at risk for lactic acidosis. And as noted above, in my case it caused digestive issues that made me strive to get off of it entirely.
Once insulin production has ceased, I would say it’s usefulness becomes more questionable, but for the LADA crowd, that could take a while.
As with everything: question everything and research/test to discover what is appropriate and helpful and what isn’t. Every person is a diffeent beast, and diabetes seems to be a case study in differentiation.
There are some Type 1’s that have a fair amount of insulin resistance so even in the case of insulin production ceasing it still may be useful.
I was diagnosed Type 2 in Nov 2010 and changed to LADA after GAD testing. I’m 45 and my endo tells me I have slow onset. So far diet and exercised have brought the numbers down, but they are creeping back up.
Ah, another LADA without insulin. So glad to know I am not the only one. Hopefully your numbers will come down again. Keep me posted.
I’m going to ask for insulin when I see my endo again in September. The glipizide seems to help to cover the meal by squeezing more insulin out of my beta cells, but can not keep up with the rest of the day unless I just don’t eat a single carb or too much of anything. Control by starvation is not a good method since I’m trying to train for an ultramarathon.