I tried my email but it said “no reply for TuDiabetes” So here it is.
> The absence of autoimmune markers and the presence of measurable insulin secretion have
> proven useful in predicting near-normoglycemic remission and long-term insulin dependence in adult patients with a history of diabetic ketoacidosis.
>
That is poorly worded. It is now well-documented that KPD’s can and do stay in remission. As to your excercise, If you read my blog then you know I use to cover 4000 to 5000 miles of cycling for better than 20 years. How can I still be diabetic. I’ve got a theory that may interest you. I ate heavy carbs during that period because I was taught to carb load. Now that I test my blood, I know that I have a severe reaction to carbs. This means my body was constantly having to pour insulin into my system to keep up with it. I now believe that I essentially shut down my pancreas by eating those high carbs. I bring this up because the few KPD’s I’ve talked to all seem to spike pretty easily with carbs. It is also known that KPD’s have problems with glucose toxicity. Hyperglycemia shuts down the ability of pancreas to secrete. This is why you went DKA even though you obviously have the ability to secrete insulin.
My point with all this is that you should think about going to a higher fat, protein diet. This keeps your body burning fat as your main fuel. You then take enough carbs to refuel your glycogen stores and you’re good. Yes, excercise is good but being a KPD means you have to aware of how you do it.
Nope, I’m not aware of DESA. I doubt, however, if they’ve ever heard of KPD T2 either.
Mike