I am a newly diagnosed diabetic at age 33. Two months ago I was in the hospital for DKA (BG 330) and my primary physicial thought I had Type 1 because there was barely any measureable amount of insulin from bloodwork before I admitted myself to the ER.
Since then, I've been placed on a Levemir and Novolog regimen. I'm currently on 47u of Levemir a day and don't have to inject Novolog unless I eat more than 45g a meal. About three weeks ago, I had my first endocrinologist visit and he immediately ordered GAD65 antibody, islet cell antibody, IA-2 antibody and C-Peptide test. All the antibody tests came back negative however my C-Peptide test was 1.3 which is very low. From this, my endo thinks that I may have Type 2 and wanted me to continue my use of insulin, diet and excercise and visit him in a couple months.
I understand that Type 2's usually produce normal amount of insulin (or close to normal) and it's just that the cells are resistant to the insulin. After 10-20 years, then the pancreas may cease to produce sufficient insulin such that the patient may need to get on insulin treatment. I seem to have skipped that phase completely and my pancreas decided to produce very little insulin. Furrthermore, it seems like my body is not that resistant to insulin since I'm only taking 47u of Levemir a day. My insulin to carb is about 1:15 if I need to use bolus for a meal that's more than 45g of carbs. My BG has been very stable since I've been out of the hospital and the lowest it ever got was 80 on a couple instances after excercising. On top of that, I thought it was very rare for Type 2's to get DKA because the amount of insulin that's in our body is enough to prevent us from going into ketoacidosis.
At this point, I'd have to believe that I'm a Type 2, however I don't feel like I have the same symptoms as other Type 2's and I'm still treating my diabetes like it's Type 1. Any thoughts?