This is interesting! It seems directly opposite to what my doctors think is going on with me: first phase is consistent; second phase is not strong enough to metabolize excess carbs (why I go high). I don’t have reactive hypoglycemia, but I do have hypoglycemia when I exercise. Apparently because first phase is still kicking out low levels of insulin, which are enough to make me go low when I get seriously glycogen-depleted.
So, this leads to a question: in Type 1, are beta cells attacked in the same way across all T1 diabetics? Or is there variation in the way beta cell dysfunction happens? I seem to have my own “basal” insulin endogenously, but am not able to produce enough to handle moderate-carb meals. So my (temporary) solution has been to lower what my beta cells are required to do by eating very-low carb (~30g-50g per day). But that sounds like others experience the opposite: a reduced level of endogenous “basal” insulin; but there pancreases can produce enough insulin in the second phase to cause a low after?
Anyhow, I find this all to be very interesting. I finally got comfortable with “where I am at in my disease progression,” in the sense of knowing that what I’m doing now is not going to work forever, but being happy it’s working for now. I still have a ton to learn about the underlying mechanisms of how it all works, though.