**i look forward to the 670g! I have been type 1 since March 1956 and have never been really under control. I have been on pumps since March 1983, which saved my life for sure, but I can go from 180 to 40 in less than 30 minutes. I control the pump, I work hard at it and feel like I never win. I am hoping that the 670g will be like playing chess against the computer, you just will never beat it. BRING IT ON, MY LIFETIME PRAYER WILL BE ANSWER!
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I don’t measure my carb grams closely as my home eating involves frequent meal repetition. I believe I’m consistently eating less than 75 grams per day with some days less than 50 grams. The trace shown above shows two conference meals, one comprised of slices ham, cheese, sliced tomatoes and a small serving of leafy greens. Dinner was one sausage, chicken, a tablespoon or two of kidney beans in a sweet barbeque sauce. I also had a table spoon of potato salad. I had two glasses of red wine over several hours, as well.
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I think the 670G will be a great fit for many people. When it shifts to its auto mode, it should really help, especially over night.
Are you in line for one or are you waiting till after the pre-orders are filled? I think I saw July for the wider release.
I spent some time this weekend with a woman who’s had one for a few weeks. She really likes it.
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i am not in line for it since I got my second 530g in March 2016. I always look down the pipeline of Medtronic to see what they are working on. I had upgraded my Revel which had 18 months left under warranty, so I figured that was perfect to get me to the 670g. Well the 530g died 19months later, so I was forced to buy a new 530G in March 2016. Since I am on Medicare they will only let you have one every five years. So my Doctor said that I needed this back in 1956, and she will fight to make it medically necessary.
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I am in line for a 670G (Priority Access). Hope to get it in June but who knows…
I’m anticipating sensor/CGM accuracy to improve over the 630G /w Enlite sensors. But I’m holding off judgement until after use it. Initially I’m planning to operate the 670G in “Manual Mode” until I get a level of confidence that sensor (CGM) accuracy is sufficient to control basal insulin administration.
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After much dithering and trepidation, mostly about not wanting to switch from G5, I’ve gotten on the 630–>670G train too. Fortunately I have a lot of G5 sensors piled up and just reordered fresh transmitters, so I’m all set to fall back on my existing methods if I decide I need to. But I’m hoping for the best, particularly if the Guardian sensors are all they’re claimed to be.
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Congratulations on your decision DrBB! There is no perfect solution integrated pump/CGM solution today, and I don’t think any of us long-term T1Ds expect that given the current technology. However it is very advantageous to have an integrated pump/CGM/meter system and the Medtronic 6 series, IMO, delivers on that I think, with limitations.
Here is a discussion where they theorize that the C peptide in the old animal based insulin provided a protective effect on Type 1 Diabetics as most of the older Type one Diabetics like you in the study had very low rates of complications. Perhaps there was a protective effect in the old insulin you were on. My grandfather was also on the pee strips and basal insulin as a type 2 and lived into his 80’s. Although he did have a few heart attacks, the final one took his life. I was also like you when I was diagnosed. I would feel hypo with any BG under 100-120. It has taken me months to where I now feel normal with a BG range of 85. I also can feel the effects of high BG when my BG hits 140 and above, when this feeling only used to hit before when my BG was 180 and over. The headache, lethargy, and brain fogginess. I’m just a type 2 so far as I know it for now, but who knows what the future has to hold. Please read that study and the comments at the bottom where they theorize that there was a definite protective effect in the old animal insulin. I was reading this because I am also worried about complications as I had high BG for a long time before my diagnosis. I also have a friend who is dying at the age of 45 because she has LADA and can have BG levels that would kill a normal person and not even feel it. She had BG’s levels in the 600’s and did not get diagnosed until she had massive complications. She has had 4 heart attacks, three complete kidney shutdowns, flatlined many times. Her doctors tell her she is indestructible as she continues to survive despite having unstable BG levels. I feel so bad for her.
Here it is: Immune From Diabetes Complications - DSM
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I also rotate five of the same meals and only have two take out options where I know the exact amount of carbs in every meal and I don’t rock the boat. It is much easier to gain good BG control like that.
Thanks for posting this link. It’s a very interesting article. I’m at 50+ years & so far my only complication is retinopathy in my left eye. So I am one of the lucky ones.
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Thanks. I thought you older Type one’s would be interested in that study. It might just be conjecture, but the numbers are there to support the theory.
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The article was not about the old animal insulin. It was just a commenter who argued that fact. It’s still an interesting read of how up to 50 percent of the Type 1 Diabetics studied had no or minimal long term complications despite not having adequate/tight control of their BG levels for many years. They think it has to do with a specific enzyme deficiency and low grade inflammation in some people. Very interesting read.
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The idea that the c-peptide left over from the production of insulin* may have a physiologic role has been around a while, and it’s intriguing. No one has yet been able to prove or disprove it conclusively, and it’s a worthy avenue for research. Something seems to have provided a degree of protection to a good number of older diabetics.
*That’s where c-peptide comes from, for anyone who doesn’t know: it’s a leftover byproduct of endogenous insulin production, and today’s artificially manufactured insulins lack it.
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@blizzard2014 and @David_dns , I participated in the Joslin Medalists Study in 2009. More than 1000 people with 50+ years of T1D participated. A very thorough examination was given at the Joslin Diabetes Center, including a c-peptide test. My c-peptide result is less than 0.1, indicating that I do not produce any of my own insulin. In an early stage of the study, with about 500 participants, 66% of them were producing an unexpected amount of insulin. That was further confirmed by a GTT. These individuals are insulin dependent, but the insulin produced in their bodies contains c-peptide. The animal insulin I used for 50 years also contained c-peptide. I asked Dr. King, head of the study, if c-peptide might explain why thousands of T1D’s have lived so long without any serious complications. He said he did not think the c-peptide was a significant factor. I am not so sure he is correct, but no other explanation has been found that explains our good health. I have not had c-peptide in my insulin for the last 20 years, and during that time, I have been diagnosed with retinopathy and neuropathy. Pumping gave me better control, and the retinopathy is no longer a problem, but the neuropathy is now bothering me a lot. I will always think that the c-peptide may have protected me for my first 50 years of T1D. Many medalists are giving very similar reports in the medalist group on Facebook.
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Richard and many other longtime T1’s feel that they got protection from the c-peptides in the older insulins. At the same time, many of their fellow T1’s had horrible complications and died while on those insulins. For sure, I don’t know. I know that the biggest improvement I had in my BG levels came with starting to use Lantus and I have never wanted to go back to Lente and NPH. But I do think that many of us have had luck when it comes to survival and relatively good health. I’m a baby compared to @Richard157 with only 40 years of T1.
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I think that the levels of insulin production found in long-term Type 1s were very, very small, though. Based on the study I read (which I’m pretty sure is the same one @Richard157 mentions), 67% of the participants had detectable levels of c-peptide, but the cut-off they used for “detectable” was anything above 0.03. Only 3.5% of participants had c-peptide levels above 0.17. So, it would seem that the differences in c-peptide exposure would be quite small even for most of those who did produce a tiny amount of their own insulin. It is an interesting idea, though. I thought I read at some point that they were doing studies on c-peptide or maybe even working on a synthetic version to inject weekly. Not sure what ever happened to that, or if I’m just imagining it.
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Hi @Jen, the group that attempted tp produce an injectable form of c-peptide halted their program due to poor results during human trials. Here is the link:
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@Laddie, there was a long discussion about c-peptide on the site diabetesforums.com. Many there did believe it helped protect us from complications, but more recently I am thinking it is having good genes more than anything else.
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Well, the jury is still out. But if I had to bet, I’d bet on heredity.
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When attending a conference I routinely use a temporary basal of +40%. Nothing to do with the meals. It corrects for the effect of sitting on my a**e in a lecture theatre for several hours. Same for long-haul air travel or anything else that involves more than an hour or two of relative inactivity.