Hi there…you sound JUST like me, only mine was caught a bit later - my post-prandials were higher and I did not have the reactive hypo’s. My IGT was caught by a routine annual physical…with a High Fasting BG of only 105. My internist immediately dubbed me Pre-Diabetic T2 - and was not particularly worried…but I WAS b/c…like you I had IDDM’s in my immediate family. My dad AND his older sister were both adult onset IDDM’s. My father became insulin dependent within 6 months of dx. His oldest sister (so 2of the for sibs ended up adult onset IDDM’s) died pof DM complications after a leg amputation…so I was hyper-concerned about a 105!
Like you…I got me a meter and tested. IMAGINE my surprise…b/c with a mere 105 I wasn’t THAT concerned…when my first testing the next a.m. after a “healthy” breakfast of 1/2 cup of steel cut oats (from scratch) with home made applesauce (very little sugar in entire batch) shot me well up over 200 1 hr PP. I like to died. Just could not believe the meter.
Thus began my journey of test, discovery of just how reactive to carbs I was…elimination of carbs, winnowing and reduction of foods from diet until (and you’ll understand this as an RD) I got to the point where I was having trouble finding enough to eat. The 1/2 of an orange bell pepper (eaten by itself, 7 carbs) that shot me to 197 at 45 minutes after I ate it (after walkign 3 miles)…that was the straw that broke the camel’s back.
I won’t bore you with all of the details, but at that point, after talking to my father’s internist (whose passed on) and reading up on LADA…I decided I needed to take action. By this time, I knew my A1C was only 5.4 and the Internist REALLY was NOT worried… kept swearing all I had was IGT…and thought I was highly anxious.
But, I fed her all my data: by this time, exercising and eating much less, I was losing about a pound every other day! So I was getting quite slender…no other signs of metabolic disorder…IDDM in family hx… and I sent all my meter readings and what I’d eliminated from my diet.
It made her head swim and she referred me to my Endo (I have Hashimoto’s as well…so had a relationship already).
The Endo downloaded the data from my meter and…saw all the readings over 200 and pronounced me officially diabetic. Sent me for GAD’s and then for a C-peptide with a High B-G reading (over 200…I go to eat oatmeal and applesauce for breakfast one more time…WITH Brown sugar!) and she declared that I was NOT insulin resistant. NOR DID I have auto-immune diabetes…
Hmmmm - quite a mystery…isn’t it… it gets better…
I’ve continued to dig.
Turns out that my great grandmother died in Russian in 1887, 5 yrs after she gave birth to my grandfather - the father of the 2 sibs who were IDDM. Okay…no records…but…she was just the right age for MODY to appear as Gestational Diabetes…and if she had as severe a case of “wasting disease” as they called it back then… there was no insulin and nothing that could have been done for her and the progression could have easily killed her when my Grandfather David was 5 yrs old. And noone would have thought to mention this to anyone who would have known it would be significant (it was all men who were left…my grandfather, his father and my granfather’s older brothers by his dad’s first wife…wives tended to die on that Greatgrandpa of mine…the first wife died in childbirth!)
The only thing I don’t understand is whether MODY can be passed on and not manifest as the disease in one generation (my grandfather himself…to the best of my knowledge did not have diabetes…but two of his children did)…or did he pass on a recessive gene and did my dad and his sister inherit another recessive copy from their mother!
I know it’s all really complicated…
Any way…Sorry to drone on and one…but it’s quite amazing to be the historical medical detective.
And FINALLY on the T3 front…wait til you see the article I just found today on a website in the U.K. on the link between the hormone T3 and Diabetes…it has to do with mice. I’ll copy the article here. Good luck!!! April
A THYROID HORMONE THAT COULD FIGHT AGAINST DIABETES
Tue, 30 Nov 2010
Researchers at the University of Oklahoma in the US have discovered a hormone in the thyroid that could help millions of people with diabetes. The scientists were actually examining the effect of cold temperatures and a thyroid hormone on cardiovascular disease, when they realised that, as coldness can induce higher blood pressure in animals and humans, diabetic mice might develop the condition quicker in cold temperatures.
On putting both diabetic mice and non-diabetic mice into a box maintained at a constant 41 degrees, it was found that although the non-diabetic mice survived, the diabetic mice died in just two hours. This breakthrough meant that the animals could not maintain their body temperatures, and made them realise that perhaps giving the diabetic mice a thyroid hormone would increase their body temperatures.
An injection of thyroid hormone, T3, did raise the body temperature and also significantly decreased blood sugar levels within a couple of hours and reduced it by over half in four hours. They also discovered that the thyroid hormone level in the diabetic animals is extremely low, while the blood sugar was about five times higher than normal levels. It is now hoped that T3 could be a potential treatment for type 2 diabetes, as the researchers try to uncover what allows the hormone to lower blood sugar levels.
Zhongjie Sun, who led the study at the OU Health Sciences Center, which will be published in the British Journal of Pharmacology, said “these are important studies and could have a significant impact for the millions of people with type 2 diabetes .”