Ok, first, this is a bit of a confusing read. You may want to edit it. If you have (four) leading spaces in a line the forum can interpret that for special formatting (called a code block). That leads to weirdness.
So let me ask you, are you really doing you dissertation on the “Etiology and Pathogenesis of Type 2 Diabetes Mellitus” or is this a section?
And as to the quote from Surampudi, I don’t have any quibble with it but it might not be the most authoritative source. A better source would be Ralph DeFronzo who has literally hundreds of pubmed citations. His 2008 Banting award lecture presented a model of eight fundamental defects associated with T2. One of those defects is the increased production of glucose by the liver but another is also a set point problem which is really a defect with the hypothalamus. DeFronzo’s paper would be considered far more authoritative. More recently Stanley Schwarz has argued for a reclassification of diabetes based on fundamental defects. He recognizes defects in a manner that is consistent with DeFronzo. His paper in Diabetes Care can be found here. There has been some discussion of Schwarz and background information here at TuD.
I appear to have some mix of the defects with my set-point and excess glucose production with my liver (I also have insulin deficiency). None of the available medications worked to correct my chronic high fasting blood sugars but once I started a basal insulin my fasting blood sugars became immediately controllable. Unfortunately I was repeatedly denied a basal insulin by the health care system and had to make my own decision to start managing my diabetes with insulin. I do hope that things change in the future.