Pumps use only fast-acting insulin ( what I believe you referred to as bolus insulin) There is basal all-day delivery of the fact-acting insulin which covers your basal or background insulin needs.
THATâS where I got confusedâŚ.I was told bolus was fast; basal was slow-acting. When speaking to the Twiist rep he kept saying âbasalâ and I thought he didnât know his termsâŚ..that pumps only use fast-acting (bolus)? Am I wrong?
Oh he was using âbasalâ as a verb (or adjective) that the fast-acting delivers all-day basal deliveryâŚ..See how uninformed I am???/
Yes! Using basal and bolus as a verb or describing the purpose of insulin.
Just wait until your spell check insists you are talking about âblousesâ
Thatâs diabetes humor! (not too much is funny about it)
Analysis of curves to adjust basels should only be done when looking at curves over a week. But you are asking about BG increasing after taking a correction bolus, but what about meal boluses? I have noticed this occurring with me over the past 4 months, after 35 years of pumping. I have not been able to figure out why. I have noticed it sometimes takes longer for a bolus to take effect dependent on the time of the day.
Does this happen on occasion, or routinely? Is heat a factor? Length of time insulin has been in the cartridge (degradation)?
Mike
Itâs more likely the food you re eating and the mix of carb fat and protein.
Also exercise plays a role as does any med that slows digestion.
My whole 40 years with type 1 has been a cycle of different things all effecting glucose in a different way.
For me fats play a big role in altering absorbtion of carbs, and led me to try the mastering diabetes, low fat diet.
I did an experiment with toast. I eat a piece of bread on its own and it requires 1.5 units to metabolize in 45 min Iâm back to normal.
Add a tablespoon of peanut butter and it takes me 3 units and nearly 3 hours to metabolize.
I rarely eat bread now and I avoid nuts because they are high in fat.
But what I learned was to separate fats and carbs.. the combo is what makes it unpredictable, And yes that opposite of every other nutrition book or way of eating has taught me over the years.
Itâs not for everyone, but my sugars are much more stable when I avoid eating fat, and I use less insulin even though I eat more carbs, 110 carbs per day on average
Does it matter if you use peanut butter without added sugar ?
Apologies still new to t2.
While I prefer Laura Scudder peanut butter that requires stiring. My wife doesnât. Herâs has sugar and palm oil, mine is peanuts and salt. They both have 7g of carbs per 2 tablespoons.
Carbs are carbs, although carbs from rice are super carbs for me,
The point of the experiment was using pure peanut butter. No salt no sugar.
It demonstrates that fats decrease insulin sensitivity.
Or, more likely, protein. For people on a keto diet protein (so no, or very little, carb energy) protein directly inputs to the Krebâs cycle. This does decrease the effect of insulin because now there is a competing energy source.
Fats can do that too but the main problem with fats is the delay; they donât decrease insulin sensitivity any more than proteins do but they are slower about it: They do catabolise into Acetyl-CoA and end up doing the same thing as proteins:
Or, for those who are not anti-wikipedia, just read the wikipedia article (which I wonât quote because Iâve been told off for quoting wikipedia in the past.)
If you want a fast way of decreasing insulin sensitivity sit back and take a drink. Ethanol zaps pretty much everything until it is catabolised away.
Iâm really leaning towards my body shifted into fasting mode trying to supply some energy based on some combination of messed up signals. Iâll have to think twice about skipping lunch when high in the future.
That strikes me as quite possible, given the whole set of weird things like âsticky highsâ and, indeed, DKA (which I guess is sort-of understood).
When high I try to pre-bolus by up to 30 minutes (limited by the amount of warning I get of incoming food). Since I use AndroidAPS I donât, most of the time, attempt to do corrections; AAPS got there first, but sometimes I do and that seems to work out ok; AAPS undoes my errors ![]()
I donât go low; my TIR âlowâ figure is 0, although sometimes the CGM says I did. It lies. If I do go low the glucagon response messes everything up for the rest of the day.
I do âskipâ meals and sometimes I add meals, so Iâm a real challenge for any algorithm. When I was paid to programme computers I pulled the plug out of the wall on my office computer before I went home. NT was meant to survive power failures and I have to admit it did, despite my evil best efforts.
Just to put a fork in this topic and call it done @MikeR1 figured it out in another topic.
For me the statin doesnât cause a rise but something more like insulin resistance. Maybe a counterregulatory response to insulin? And itâs all my fault, I stated on 20mg and my endo said great, done. I asked for more dreaming of a LDL in the 40âs. Over a year I worked my way up to 80mg, thinking there were no side effects. My LDL never got lower than the mid 60s, was pretty consistently 78ish pre-statin. I stopped and the resistance faded away. Going to experiment further to see if it was just a dose problem.
This review paper shows this is not just your experience:
The paper (beyond the summary) also suggests that âstatinsâ vary in bad side effects, or, as I would put it, âThere is no such thing as a statin.â I.e. âstatinâ is an effect, not a treatment or a chemical for that matter.
Yeah, ok, I didnât read past page 2 but I established it was well written, balanced and therefore worthy of consideration. I invite anyone who cares to refute the summary to testify or to confound the information on the basis of who paid the certainly well paid authors.
Itâs easy to find other references. Statins cause T2. The cause seems to be because of decreased insulin sensitivity, a known effect of statins.
OMG, I spent all those years refusing statins offered on the basis that I was a diabetic and statins are recommended for diabetics. AARGH.
I do remember one thing I learned from my mother. She was a nurse, a midwife and later a somewhat frustrated administrator in an old-peoplesâ home. She habitually refused all medical treatment until after she died as a result (annoying everyone). A person I greatly respect.
